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custom targeted gene panel  (Twist Bioscience)


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    Twist Bioscience custom targeted gene panel
    Custom Targeted Gene Panel, supplied by Twist Bioscience, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/custom+targeted+gene+panel/Custom+Panel/pm42123485-258-4-8
    Average 98 stars, based on 1 article reviews
    custom targeted gene panel - by Bioz Stars, 2026-11
    98/100 stars

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    other:

    Article Title: Type 1 Brugada Pattern Triggered by Low-Grade Fever: Implications for Diagnosis and Risk Stratification
    Article Snippet: For library preparation, a custom targeted gene panel (Twist Custom Panel kit from Twist Bioscience, San Francisco, CA, USA) was used that examines the coding regions—including exon/intron boundaries ±20 nucleotides—of 43 genes related to cardiac disorders, such as cardiomyopathy ( ACTC1 , ALPK3 , BAG3 , DES , DSC2 , DSG2 , DSP , FLNC , JUP , LMNA , MYBPC3 , MYH7 , MYL2 , MYL3 , PKP2 , PLN , RBM20 , SCN5A , TMEM43 , TNNC1 , TNNI3 , TNNT2 , TPM1 , TTN ); long-QT syndrome ( CALM1 , CALM2 , CALM3 , KCNH2 , KCNQ1 , TRDN ); and PAH—pulmonary artery hypertension ( ABCC8 , ACVRL1 , AQP1 , ATP13A3 , BMPR2 , CAV1 , EIF2AK4 , ENG , GDF2 , KCNK3 , SMAD9 , SOX17 , TBX4 ).

    Article Title: Type 1 Brugada Pattern Triggered by Low-Grade Fever: Implications for Diagnosis and Risk Stratification.
    Article Snippet: ECGs were recorded at 25 mm/sec recording speed (Hewlett Packard Page Writer 200i, Hewlett-Packard Company (HP), 1939, Palo Alto, CA, USA) while the patient was in the supine position, breathing freely and not talking.

    Article Title: Type 1 Brugada Pattern Triggered by Low-Grade Fever: Implications for Diagnosis and Risk Stratification.
    Article Snippet: For library preparation, a custom targeted gene panel (Twist Custom Panel kit from Twist Bioscience, San Francisco, CA, USA) was used that examines the coding regions—including exon/intron boundaries ±20 nucleotides—of 43 genes related to cardiac disorders, such as cardiomyopathy (ACTC1, ALPK3, BAG3, DES, DSC2, DSG2, DSP, FLNC, JUP, LMNA, MYBPC3, MYH7, MYL2, MYL3, PKP2, PLN, RBM20, SCN5A, TMEM43, TNNC1, TNNI3, TNNT2, TPM1, TTN); long-QT syndrome (CALM1, CALM2, CALM3, KCNH2, KCNQ1, TRDN); and PAH—pulmonary artery hypertension (ABCC8, ACVRL1, AQP1, ATP13A3, BMPR2, CAV1, EIF2AK4, ENG, GDF2, KCNK3, SMAD9, SOX17, TBX4).



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    Fig.1 Flow diagram of the study design for NGS analysis. NGS, next-generation sequencing; OPMDs, oral potentially malignant disor- ders

    Journal: Discover oncology

    Article Title: Oral cancer driver gene mutations in oral potentially malignant disorders: clinical significance and diagnostic implications.

    doi: 10.1007/s12672-025-01923-7

    Figure Lengend Snippet: Fig.1 Flow diagram of the study design for NGS analysis. NGS, next-generation sequencing; OPMDs, oral potentially malignant disor- ders

    Article Snippet: Thus, we developed a custom target gene sequencing panel (AmpliSeq for Illumina Customs DNA Panel) capable of detecting full-length mutations in TP53, NOTCH1, CDKN2A, FBXW7, as well as mutations in HRAS, PIK3CA, and BRAF.

    Techniques: Next-Generation Sequencing

    Fig. 5 OLP case with ODGM. a–d Case with ODGM (case 15). a At the initial presentation, we observed non-removable white patches with mottled appearance and surrounding redness, leading to a clinical diagnosis of OLP. b A biopsy confirmed the histopathological diagnosis of OLP. c Eleven months after the initial presentation, a thickened white patch was observed on the posterior buccal mucosa. d A second biopsy led to a histopathological diagnosis of OED. e, f OLP case without ODGMs (Case 24). e Oral cavity image. f H&E staining. g Timeline of clinical course for case 15. ODGM, oral cancer driver gene mutation; OED, oral epithelial dysplasia; OLP, oral lichen planus; NGS, next- generation sequencing

    Journal: Discover oncology

    Article Title: Oral cancer driver gene mutations in oral potentially malignant disorders: clinical significance and diagnostic implications.

    doi: 10.1007/s12672-025-01923-7

    Figure Lengend Snippet: Fig. 5 OLP case with ODGM. a–d Case with ODGM (case 15). a At the initial presentation, we observed non-removable white patches with mottled appearance and surrounding redness, leading to a clinical diagnosis of OLP. b A biopsy confirmed the histopathological diagnosis of OLP. c Eleven months after the initial presentation, a thickened white patch was observed on the posterior buccal mucosa. d A second biopsy led to a histopathological diagnosis of OED. e, f OLP case without ODGMs (Case 24). e Oral cavity image. f H&E staining. g Timeline of clinical course for case 15. ODGM, oral cancer driver gene mutation; OED, oral epithelial dysplasia; OLP, oral lichen planus; NGS, next- generation sequencing

    Article Snippet: Thus, we developed a custom target gene sequencing panel (AmpliSeq for Illumina Customs DNA Panel) capable of detecting full-length mutations in TP53, NOTCH1, CDKN2A, FBXW7, as well as mutations in HRAS, PIK3CA, and BRAF.

    Techniques: Biomarker Discovery, Staining, Mutagenesis, Next-Generation Sequencing